Meseguer 2024 — Trends in Cause-Specific Mortality by Race and Hispanic Origin, 1999–2019

mortalityrace-ethnicitypublic-healthdeaths-of-despairopioidscirculatory-diseasecancerdemographysocial-security

Summary

Purely descriptive analysis of age-adjusted cause-specific mortality trends 1999–2019 for four race/Hispanic-origin (RE) groups — White non-Hispanic (WNH), Hispanic, Black, and Asian/Pacific Islander (API) — using Centers for Disease Control and Prevention (CDC) WONDER administrative death records and International Classification of Diseases, 10th Revision (ICD-10) classifications. All-cause mortality improved for all groups but the improvement was front-loaded to 1999–2009; after 2010, midlife mortality (ages 25–64) stalled and then reversed for nearly every subgroup. The paper catalogs the explosion of deaths of despair (poisoning, suicide, alcohol) across RE groups, the persistence of large Black-WNH mortality disparities despite narrowing, the Hispanic mortality paradox, and the relevance of these RE-group mortality differentials to Social Security program outcomes.

Key Claims

Concepts Introduced or Extended

Entities Mentioned

Quotes

"Mortality differentials by RE group and sex affect Social Security retirement and disability program-participation outcomes."

"The Hispanic population has a lower all-cause mortality rate than WNH people, a phenomenon often referred to as the Hispanic mortality paradox, given the WNH community's advantages in socioeconomic status."

"The growth in opioid-involved overdose deaths in the Black community now outpaces that of any other RE group, as fentanyl and other synthetic opioids disproportionately affect overdose death rates among older people in the Black community."

"In 1999, the all-ages death rate from infectious and parasitic diseases for Black males (75.9) was more than 3.5 times that of WNH males (20.8) ... By 2019, the mortality rate of Black males at all ages had declined by more than half."

My Take

A careful, comprehensive descriptive atlas — not a causal analysis. Its value is as a data synthesis rather than an identification exercise: it pulls the ICD-10 cause-of-death universe across four RE groups and 20 years into one place with careful age-adjustment. Three things stand out. First, the racial universalization of deaths of despair is documented more granularly here than anywhere else — the poisoning data by RE group and age are striking; the fentanyl wave's movement into the Black community (the 2019 crossover in poisoning mortality) is underreported. Second, the respiratory disease exception (WNH worse than Black at older ages) is underappreciated and points to COPD's smoking-history dependence. Third, the American Indian/Alaska Native (AIAN) exclusion is an important limitation: the group with the worst mortality trajectory is removed from the analysis on data-quality grounds, likely understating the full severity of U.S. mortality disparities. The paper is the natural complement to Woolf and Schoomaker (2019) — Woolf provides the 60-year joinpoint narrative; Meseguer provides the RE-stratified cause decomposition for the most recent two decades.